Seminars in Nephrology
Volume 28, Issue 4 , Pages 409-415, July 2008

Viral Hemorrhagic Fever–Induced Acute Kidney Injury

  • Emerson Q. Lima, MD, PhD

      Affiliations

    • Division of Nephrology, Sao Jose do Rio Preto Medical School, Sao Jose do Rio Preto, Sao Paulo, Brazil.
    • Corresponding Author InformationAddress reprint requests to Emerson Q. Lima, MD, PhD, Av. Brigadeiro Faria Lima 5416, São Jose do Rio Preto, Sao Paulo, Brazil 15090-000.
  • ,
  • Mauricio L. Nogueira, MD, PhD

      Affiliations

    • Laboratory of Virology, Division of Infectious Diseases, Sao Jose do Rio Preto Medical School, Sao Jose do Rio Preto, Sao Paulo, Brazil.

Summary 

Viral hemorrhagic fevers (VHFs) are diseases caused by the RNA virus from 4 different families (Flaviridiae, Arenaviridae, Bunyaviridae, and Filoviridae) that are acquired through the bite of an infected arthropod or by the inhalation of particles of rodent excreta. Among the VHFs, dengue and yellow fever are the most prevalent in tropical regions worldwide. The clinical presentation is characterized by fever, malaise, increased vascular permeability, and coagulation defects that can result in bleeding. Acute kidney injury is an uncommon complication but renal dysfunction has been associated with various VHFs. In this article we review the renal manifestations of dengue and yellow fever infections.

Keywords: Acute kidney injury, viral hemorrhagic fever, dengue, yellow fever

To access this article, please choose from the options below

Login to an existing account or Register a new account.

  • Purchase this article for 31.50 USD (You must login/register to purchase this article)

    Online access for 24 hours. The PDF version can be downloaded as your permanent record.

  • Subscribe to this title

    Get unlimited online access to this article and all other articles in this title 24/7 for one year.

  • Claim access now

    For current subscribers with Society Membership or Account Number.

  • Visit SciVerse ScienceDirect to see if you have access via your institution.
 

 Emerson Q. Lima was supported in part by a grant from Fundação de Amparo à Pesquisa do Estado de Säo Paulo (FAPESP), Brazil; and Mauricio L. Nogueira was supported by FAPESP and Conselho Nacional de Desenvolvimento Científico e Tecnológico grants.

PII: S0270-9295(08)00087-9

doi:10.1016/j.semnephrol.2008.04.009

Seminars in Nephrology
Volume 28, Issue 4 , Pages 409-415, July 2008